Nutritional management of obesity in dogs |
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Current estimates of obesity in dogs suggest a prevalence of around 20-40% in various countries1. While some diseases (such as hypothyroidism and hyperadrenocorticism) predispose to obesity,
in most cases the underlying cause is a mismatch between energy intake and energy expenditure.
A wide variety of diseases are associated with obesity in dogs1,2. Importantly, Nestlé PURINA recently
completed a lifelong study of dogs that provided crucial information showing excess bodyweight
is detrimental to their longevity as well – in that study even moderately overweight dogs were at
greater risk for earlier morbidity and a shortened lifespan3.
The management of obesity in dogs usually combines a weight loss diet with increasing the daily
exercise routine, and perhaps feeding two meals per day with a reduced number of treats, may be
the most effective means of managing obesity. There is no doubt that obesity clinics can also be a
valuable management tool by:
- Helping to maintain motivation and compliance of the pet owner4
- Enabling careful monitoring of the response of the individual
- Providing an opportunity to make any changes necessary to the management regime
- Reducing the likelihood of rebound weight gain
NUTRITIONAL MANAGEMENT OF DIABETES MELLITUS IN DOGS

The most common underlying cause of diabetes mellitus in dogs is immune-mediated 'type I'
diabetes, evidenced by the presence of antibodies against ß-cells5. Acute or chronic pancreatitis is also important and may be present in up to 35-40% of cases5. The primary goal of therapy is to maintain blood glucose concentrations as close to normal as possible. Additional goals include achieving
and maintaining normal body weight (as obesity contributes to insulin resistance), and managing
complications of diabetes such as hypercholesterolaemia and other lipid changes, and oxidative stress.
Optimal management combines insulin therapy, appropriate exercise and nutritional therapy.
The following dietary characteristics are recommended to help manage the disease6-8:
- A diet low in simple carbohydrates to help reduce post-prandial hyperglycaemia.
- A diet high in complex carbohydrates that will help delay glucose production and absorption
and thus reduce post-prandial hyperglycaemia.
- Both soluble and insoluble fibre appear to help reduce post-prandial hyperglycaemia.
- Mixed fibres (maize, barley, soya, pea fibre) are better at controlling post-prandial hyperglycaemia
in dogs than insoluble fibre (cellulose) alone9.
- Fibre addition is also beneficial in controlling hypercholestrolaemia.
- Controlled fat levels help to maintain an ideal body weight, prevent obesity-associated insulin
resistance, and may help reduce diabetes-associated hyperlipidaemia.
Effect of a diet low in simple carbohydrates and high in complex carbohydrates on the blood glucose
concentration after feeding.
CLINICAL ADVANTAGES WITH THE USE OF CANINE OM

Canine OM provides:
- An increased protein level and a higher protein to calorie ratio, proven to preserve lean body mass during weight loss5. Dogs should be fed to encourage gradual weight loss with preservation of lean body mass, which reduces the risk of rebound weight gain.
In weight loss studies, increased dietary protein spared lean body mass and facilitated greater fat loss in dogs during weight loss.>
- A high nutrient:energy ratio to compensate for the reduced calorie intake.
- A low fat level, which helps lower serum cholesterol levels and control hypercholesterolaemia
and hyperlipidaemia.
- Increased fibre to help improve satiety and reduce overall caloric intake.
- Glucoregulation through a combination of high levels of complex carbohydrates and mixed fibre
sources, combined with low levels of simple sugars.
- Additional benefits such as enhanced vitamin E concentrations to combat the oxidative stress
associated with diabetes and/or obesity.
1. German AJ (2006) The growing problem of obesity in dogs and cats.
J Nutr 136: 1940S-1946S.
2. Laflamme D (2006) Understanding and manageing obesity in dogs and cats.
Vet Clin Small Anim 36: 1283-1295.
3. Kealy RD, et al. Effects of diet restriction on life span and age-related changes in dogs.
J Am Vet Med Assoc 2002; 220:1315-1320.
4. Bland IM et al. (2010) Dog obesity: veterinary practices' and owners' opinions on cause and management.
Prev Vet Med 94: 310-315.
5. Hannah SS, Laflamme DP. Increased Dietary Protein Spares Lean Body Mass during Weight Loss in Dogs.
J Vet Int Med. 1998; 12:224.
6. Rand JS et al Canine and feline diabetes mellitus: Nature or nurture.
J. Nutr. 2004: 134; 2072S-2080S
7. Jackson, JR, Laflamme DP (1996) Effects of diets on post-prandial blood glucose in dogs. Purina Nutrition Forum proceedings.
8. Nelson RW, et al.(1991) Effects of dietary fiber supplementations on glycaemic control in dogs with alloxaninduced diabetes mellitus. Am J Vet Res; 52:2060-2066.
9. Graham PA, et al. (1994) Canned high fiber diet and postprandial glycemia in dogs with naturally-occurring diabetes mellitus. J Nutr; 124: 2712S-2715S
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